Coagulation-pathway_stable.xml 763 KB
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<?xml version="1.0" encoding="UTF-8"?>
<sbml xmlns="http://www.sbml.org/sbml/level2/version4" xmlns:celldesigner="http://www.sbml.org/2001/ns/celldesigner" level="2" version="4">
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<model metaid="RAS_complete2" id="Coagulation_goar">
<notes>
<html xmlns="http://www.w3.org/1999/xhtml">
<head>
<title/>
</head>
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<body>Authors:
<br/>
Goar Frishman, Helmholtz Zentrum München /Institute of Experimental Genetics, Munich, Germany
<br/>
<br/>
Gisela Fobo, Helmholtz Zentrum München / Institute of Experimental Genetics, Munich, Germany
<br/>
<br/>
Corinna Montrone, Helmholtz Zentrum München / Institute of Experimental Genetics, Munich, Germany
<br/>
<br/>
Marek Ostaszewski, Luxembourg Centre for Systems Biomedicine (LCSB), Luxembourg
<br/>
<br/>
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Description: Thrombosis model of COVID-19
</body>
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</notes>
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<annotation>
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<celldesigner:modelDisplay sizeX="7000" sizeY="20000"/>
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<celldesigner:listOfIncludedSpecies>
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<celldesigner:listOfStructuralStates>
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<body>
Ang I can be cleaved by NEP to Ang (1–7).
ACE and NEP hydrolysed Ang I efficiently, whereas ACE2 hydrolysed Ang I only very slowly.
PMID:15283675
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<celldesigner:notes>
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<body>
Long Name:  FibrinogenSynonym:  Factor IAccession:  P00406
</body>
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<celldesigner:notes>
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<body>
Long Name:  FibrinogenSynonym:  Factor IAccession:  P00406
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</html>
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<body>
Long Name:  FibrinogenSynonym:  Factor IAccession:  P00406
</body>
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</html>
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